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Findings include: Suppression of metastatic genes – 70% reduction in genes overexpressed in metastatic colon cancer Enhanced DNA repair gene expression supporting genomic stability Activation of tumor suppressor pathways including p53-related genes Anti-angiogenic effects in tumor models through balanced VEGF modulation Reduced cancer cell migration in multiple cell line studies [CALLOUT BOX – Highlighted] Critical Research Limitation: Despite extensive preclinical research spanning five decades, GHK-Cu has limited published human clinical trial data

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